By using this analysis in the HIV+ individuals, we observed that expression of IL-32D and IL-32θ, IL-18, VEGF-A and IFNβ remained significantly associated with coronary artery atherosclerosis independently of age, smoking or statin treatment (p=0.031, p=0.042, p=0.001, p=0.029 and p=0.046, respectively), whereas TRAIL, IL-1β and FLT3L showed borderline significance (p=0.052, p=0.055, and 0.059, respectively). Here, FLT3LG is linked to coronary atherosclerosis.