In a recent re-analysis of leprosy public microarray datasets, mainly comparing skin lesions from different clinical forms, we confirmed previous genes and pathways corroborating the predominance of cellular immunity, leukocyte differentiation, vitamin D receptor (VDR)-mediated microbicidal responses, and granuloma formation at the T-pole, while the L-pole exhibited scavenger receptors and lipid metabolism genes (14). Here, VDR is linked to leprosy.