The IPA (Figure 2) showed that “Atherosclerosis Signaling” and multiple IL-17-related pathways (“Role of IL-17F in Allergic Inflammatory Airway Diseases,” “Role of IL-17A in Psoriasis,” “Differential Regulation of Cytokine Production in Macrophages and T Helper Cells by IL-17A and IL-17F”) were significantly activated in the AT group, while multiple metabolic synthesis-related pathways (“Histamine Biosynthesis,” “Putrescine Biosynthesis III,” “Catecholamine Biosynthesis,” and “Prostanoid Biosynthesis”) were inhibited. This evidence concerns the gene IL17F and psoriasis.