TAC1 and acute respiratory distress syndrome: The interaction of the H1N1-M2 protein (IAV A/PR/8/34 strain) with pulmonary cell NKA modifies the classic profile of the NKA α1 subunit’s basolateral expression and disperses the β1 subunit, worsening edema clearance during acute respiratory distress syndrome (ARDS)-induced by H1N1(IAV A/PR/8/34 strain) (Peteranderl et al., 2016).