IL1B and Alzheimer disease: Because of its ability to induce on its own or to work synergistically with IL1β and IFNγ to induce the expression of other pro-inflammatory cytokines (Korn et al., 2009; Meares et al., 2012; Zimmermann et al., 2018), IL-17A plays a prominent role in the induction of the neuroinflammation implicated in the onset of different hallmarks of AD, including the increase in Aβ load.