Compared with previous studies, the proteomics of orbital tissues in TAO revealed that many myosins are significantly upregulated, [6] such as MYH2 (3.5‐fold), MYH6 (2.5‐fold), and MYH3 (2.3‐fold), as well as myosin heavy chain 11 (MYH11) in our proteomic data. The gene discussed is MYH6; the disease is thromboangiitis obliterans.