FCGR2A and thromboangiitis obliterans: Barrera et al. [14] reported that FCGR2A and FCGR3A might be more susceptible to rheumatoid arthritis (RA), which implied that a vigorous inflammatory response might induce an increase in Fc gamma receptors, which is in agreement with a previous study that reported proteins involved in inflammatory processes are upregulated in TAO [8].