PTPA and pulmonary fibrosis: Sun et al. reported that mice with myeloid-specific deletion of PP2Acα developed more exuberant lung fibrosis in response to bleomycin [18], while Hou et al. and Deng et al. reported that targeting PP2A retarded kidney fibrosis, suggesting a pro-fibrotic role for PP2A in kidney diseases [23, 24].