Although most anti‐HMGCR‐positive patients in the study showed DM/DM‐like skin lesions, the histological pattern was not classic for typical DM pathological features as none of perifascicular atrophy, MxA‐positive fibers, perifascicular expression of MHC‐I, or obvious MAC‐positive capillary was detected. The gene discussed is HMGCR; the disease is dermatomyositis.