FOXA1 and necrotizing enterocolitis: In our recent series of studies on expressional arrays of NEC tissues, we identified aberrant mRNA expression patterns, which suggested a complex mechanism that was initiated at receptor TLR4, mediated via key transcription factors NFKB2, AP‐1/FOSL1, FOXA1, and HIF1A, and extended to downstream pathways involving angiogenesis, arginine metabolism, cell adhesion and chemotaxis, extracellular matrix remodeling, hypoxia/oxidative stress, inflammation, and muscular functions [7].