The results showed that BACH1 knockdown significantly inhibited the density of CD31+ blood vessels and VEGFC expression in xenograft tumor tissues derived from mice subcutaneously injected with KYSE170‐shBACH1 cells compared with those derived from mice injected with control KYSE170‐shCtrl cells (Figure 6I and 6J). The gene discussed is VEGFC; the disease is neoplasm.