The results showed that knockdown of BACH1 significantly attenuated the tumor growth because the tumors derived from KYSE170‐shBACH1 cells were smaller than the tumors derived from the control cells (P < 0.01) (Figure 6A‐6C); moreover, overexpression of BACH1 enhanced the tumor growth because the tumors derived from KYSE150‐BACH1 cells were larger than the tumors derived from the control cells (P < 0.01) (Figure 6D‐6F). Here, BACH1 is linked to neoplasm.