At a group-wise level, patients with COVID-19 display elevated levels of brain-injury biomarkers such as glial fibrillary acidic protein (GFAP) and neurofilament light chain (NfL) in a severity-dependent manner, both in the presence and absence of neurological symptoms, highlighting that structural neurological damage occurs even in comparatively mild disease [4–6]. The gene discussed is GFAP; the disease is COVID-19.