Our study indicated that AL could significantly inhibit growth of cervical cancer cells, promote apoptosis (Fig. 1), inhibit cell migration and invasion (Fig. 2), and significantly inhibit expression of genes associated with EMT (N-cadherin, vimentin, and β-catenin) and extracellular matrix degradation (MMP-2, MMP-9, MMP-3, and MMP-13). This evidence concerns the gene CDH2 and cervical cancer.