Similarly, comparison with other datasets such as the DICE data (Fig. 3A, B) and CeD-patient derived CD4+ T cells from PBMCs described by Quinn et al. (data not shown), validate our findings that the gsTcells have a similar but distinct expression profile and that important cytokines and other genes are associated to CeD etiology, such as IFNG, IL21, IL17A and IL454, are differentially expressed. Here, IL17A is linked to cranioectodermal dysplasia.