Supporting our hypothesis of hsa-miR-29c-5p being important for establishing the ER-positive/luminal breast cancer phenotype by targeting DNMT3A which leads to hypomethylation of CpGs at ER-associated TFBSs, Chou et al. [77] found that GATA3 acts as a TF inducing the expression of the miR-29 family. This evidence concerns the gene GATA3 and breast carcinoma.