Persons with cystic fibrosis (CF), a hereditary disease caused by mutations in the genes specifying the cystic fibrosis transmembrane conductance regulator (CFTR), a chloride channel on epithelial cells [1,2,3], frequently suffer from bacterial, fungal, and viral co-morbidities of the lung that aggravate the course of disease. The gene discussed is CFTR; the disease is cystic fibrosis.