In contrast, Jy et al. found no difference in the levels of endothelial EVs (CD31+/CD42-) between aPL+ thrombotic patients and asymptomatic aPL+ patients [66], suggesting that the release of EVs is more related to the autoimmune process involving the presence of aPL than to thrombosis itself. Here, FASLG is linked to deep vein thrombosis.