It is well known that, in MM cells, lncRNA MALAT1 targeting miRNA-509–5p could control the expression of Forkhead box protein P1 (FOXP1) to stimulate the progression of MM [134], and in vivo analyses performed on male athymic BALB/c mice, 5 weeks old, demonstrated that increased lncRNA MALAT1 in blood samples of MM subjects is correlated with an adverse outcome [182,183,184]. The gene discussed is MALAT1; the disease is Miyoshi myopathy.