Peripheral blood eosinophils from patients with hypereosinophilic diseases (eosinophilic pneumonia, bronchial asthma, angiolymphoid hyperplasia with eosinophilia, and hypereosinophilic syndrome) have strong immunoreactivity for Gal-9 in the plasma membrane and cytoplasm compared to nonatopic control cells [22]. This evidence concerns the gene LGALS9 and eosinophilic pneumonia.