TLR4 and colitis: Dietary intake of βGl and βGh diminished colitis by the time-dependent modulation of autophagy and apoptosis, which involved TLRs (TLR4, TLR5) and Dectin-1 receptor activation, with βGI having a stronger effect on apoptosis (Caspase 3 expression) and βGh on autophagy (LC3B expression) [49].