IFNB1 and infection: As this upregulation was also mimicked by the stimulation of cells with IFN-β and infection with the RNA virus, Zika virus (Figure 4 and Figure 5), we hypothesise that this could be a perpetuating event, where mean LD speed and displacement could be upregulated via activation of innate immune sensors, and then kept upregulated via the production of cellular IFN.