The down-regulation of miR-19b is also observed to account for the down-regulation of low-density lipoprotein receptor (LDLR) (2.976-fold change, p = 0.024), which was significantly enriched in cellular response to organic substance term, cellular response to oxygen-containing compound term, cellular response to acid chemical term, inflammatory response term and toxoplasmosis pathway and can develop inflammatory atherosclerosis [55]. This evidence concerns the gene LDLR and toxoplasmosis.