Moreover, intramuscular and/or intravenous injection of miR-29 in mdx mice, the most widely used animal model for DMD research, promotes muscle function and inhibits fibrogenesis by targeting extracellular matrix components such as collagens and microfibrillar-associated protein 5 (Mfap5) [129,130] (Table 3). The gene discussed is MFAP5; the disease is Duchenne muscular dystrophy.