The role of SLC17A6 is less clear, however, SLC17A6 is highly expressed during early development in subcortical structures [67,68], and age-impaired rats demonstrate increased levels of SLC17A6 as compared to younger animals, with increased levels of SLC17A6 related to cognitive deficit and lower expression of glutamatergic receptors [69]. The gene discussed is SLC17A6; the disease is Cognitive impairment.