NFKB1 and cancer: AEG-1 expression is induced by lipopolysaccharide (LPS) and inflammatory cytokines, such as IL-1β and TNF-α, via the activation of NF-κB, a mechanism that might contribute to AEG-1 overexpression in cancers generated as a consequence of chronic inflammation, such as HCC and gastric cancer [130,152,153].