Moreover, a pan-cancer analysis showed the same tendency of NEAT1 to be overexpressed in various types of cancer, besides PCa, such as stomach adenocarcinoma, hepatocellular liver carcinoma, kidney papillary cell carcinoma, and kidney clear cell carcinoma, although some contradictory evidence exists regarding its tumor suppressor role in promyelocytic leukemia [33,34]. This evidence concerns the gene NEAT1 and posterior cortical atrophy.