These cells can release IFN-γ, and although phenotypically characterized by surface expression of CCR6, a marker of Th17 cells [39], they have been demonstrated to play a key role in the development of autoimmune diseases and pulmonary sarcoidosis, where their enhancement in BAL has been correlated with the onset of chronic disease [40,41]. Here, IFNG is linked to pulmonary sarcoidosis.