The development of nephrotic-range proteinuria in SLE without peripheral immune aggregate deposition or endocapillary proliferation on renal biopsy is a manifestation of SLE than the coexistence of idiopathic minimal-change glomerulopathy and SLE [20] with concomitant apolipoprotein L1 (APOL1) nephropathy [21]. The gene discussed is APOL1; the disease is Nephropathy.