Six patients (2.9%) in our cohort had IgA-nephropathy, 2 (0.9%) glomerulonephritis related to ANCA-associated vasculitis (AAV), 7 (3.4%) immune complex-mediated (other than IgA) glomerulonephritides, five advanced diabetic nephropathies (2.4%), 15 tubulointerstitial nephritides ([TIN]; 7.3%) of different nature (i.e., xanthogranulomatous or chronic interstitial nephritides, 4 of them locally developing pseudo-tumorous lesions, or of undetermined etiology), and 4 (1.9%) hypertensive nephropathy. This evidence concerns the gene CD79A and IgA glomerulonephritis.