In minimally adjusted model 1 (energy adjusted and stratified by sex, center and age), each of the three AGEs was inversely associated with the risk of HCC, with HR corresponding to 0.76 (95% CI, 0.67‐0.85), 0.76 (95% CI, 0.67‐0.86), and 0.70 (95% CI, 0.62‐0.79) per 1 SD increment in dietary intake of CML, CEL and MG‐H1, respectively (Table 2). This evidence concerns the gene CEL and hepatocellular carcinoma.