NR2F2 and endometriosis: Apart from maintaining prolonged ESR1 activity and stability, suppression of COUP-TFII levels is linked to increased proinflammatory cytokine levels (Li et al., 2013; Lin et al., 2014), angiogenesis (Fu et al., 2018), and local estrogen synthesis (Zeitoun et al., 1999; Zeitoun and Bulun, 1999; Attar et al., 2009), all of which markedly promote the pathogenesis of endometriosis.