Temporary occlusion decreases the stimulation of hypoxia-inducible factors and the release of vascular endothelial growth factor, a promoter of neoangiogenesis, tumor proliferation, and metastatic growth (Li et al., 2004), making DSM-TACE a powerful tool among therapeutic strategies to control cancer, targeting the tumor stromal environment in combination with classic chemotherapeutic agents. This evidence concerns the gene VEGFA and neoplasm.