Higher infection rates in astrocytes compared to neurons can be attributed, in part, to the markedly different kinetics of the immune response triggered by different ZIKV strains (Hamel et al., 2017) but also to the expression levels of ZIKV receptors, including the tyrosine-protein kinase receptor Axl, a type of Tyro3, DC-SIGN, and T cell immunoglobulin mucin domain-1 (Lee et al., 2018), in contrast to neural progenitor cells and neurons in which Axl is poorly expressed (Nowakowski et al., 2016). The gene discussed is AXL; the disease is infection.