Inactivation of some tumor suppressors, such as tumor protein p53 (p53) and BRCA1 associated protein-1 (BAP1), promotes tumor development at least partly via suppressing tumor ferroptosis (Jiang et al., 2015; Zhang et al., 2018; Zhang et al., 2019b; Stockwell et al., 2020). This evidence concerns the gene BAP1 and neoplasm.