Other viruses have also been associated with QTc prolongation, including HIV,13 HCV coinfection,14 and West Nile virus.15 HIV-associated inflammation, specifically elevated IL-6 level, is further independently associated with QT prolongation23 and prolonged repolarization represented as T wave onset-to-peak duration.24 Infection with SARS-CoV-2 may result in cardiac enzyme release, which parallels the systemic inflammatory response marked by elevation of ferritin, LDH, CRP, and IL-6.25 Here, CRP is linked to infection.