Native kidney disease in the clinical files included IgA in 18 cases, other glomerulonephritis (GN) in 31, hypertensive nephropathy (NP) in 25, autosomal dominant polycystic kidney disease in 28, diabetic NP in 12, congenital renal dysplasia in 10, vesico-ureteral reflux in five, amyloidosis and focal-segmental glomerulosclerosis each in three, interstitial nephritis in two and hemolytic uremic syndrome, nephronophthisis, calcineurin-inhibitor toxicity and familial mediteranean fever in one each and was unknown in 62 patients. This evidence concerns the gene CD79A and hemolytic-uremic syndrome.