BM evaluation performed at another Institution was diagnostic of AML with 46,XY,t(9;22)(q34;q11)[9]/47,idem,+8[7]/46,XY[4]; a BCR-ABL1 rearrangement (p210 fusion) (Fig. 5A) and NPM1 mutation type B were detected (Fig. 5B), whilst FLT3 was wild type. This evidence concerns the gene FLT3 and acute myeloid leukemia.