This is consistent with findings demonstrating that NRP1‐deficient myeloid cells are more pro‐inflammatory, classically activated cells in models of obesity (Wilson et al,2018), tumor growth (Casazza et al,2013; Roy et al,2017; Miyauchi et al,2018; Chen et al,2019), and sepsis (Dai et al,2017). This evidence concerns the gene NRP1 and neoplasm.