ABI3 and colorectal carcinoma: VP1::LEC2-B3 and VP1::FUS3-B3 caused strong activation of CRC in the absence ABA signaling (Figure 3F, red bars), whereas CRC activation by VP1::ABI3-B3 and VP1::VAL1-B3 transgenes showed strict ABA dependence (<0.1 CRC RNA copy per ng total RNA, except in 2 seedlings with very high VP1::VAL1-B3 expression).