These cells have clear fibrogenic properties (17–19), with high levels of intrahepatic Th17 and IL-17 commonly observed in liver fibrosis caused by various etiologies, such as HBV (20), HCV (21), cholestatic liver injury (22), autoimmune hepatitis (23), and NAFLD (24). Here, IL17A is linked to metabolic dysfunction-associated steatotic liver disease.