Clinical evidence has proposed that the cytokine storm in COVID-19 involves a vicious cycle of inflammatory responses which are characterised by excessive release of pro-inflammatory cytokines including interleukin-1 (IL-1), interleukin-6 (IL-6), interleukin-12 (IL-12), interferon gamma (IFNγ) and tumour necrosis factor (TNFα) which target lung tissues [6, 9, 10]. Here, IFNG is linked to COVID-19.