The same group elucidated that GliPR1 appears to regulate glioma cell migration and invasion via interacting with the actin polymerization regulator N-WASP and its association with the ribonucleoprotein hnRNPK [11, 25], and to promote mesenchymal transformation via a STAT3/IL-6-dependent positive feedback loop [24]. The gene discussed is STAT3; the disease is glioma.