The loss of Erbin, a ErbB2 (v-erb-b2 avian erythroblastic leukemia viral oncogene homolog 2) interacting protein activity in cervical cancer, was reported to upsurge STAT3 phosphorylation, thus facilitating anoikis resistance and tumor invasiveness in vitro and in vivo (83). This evidence concerns the gene STAT3 and neoplasm.