The loss of Erbin, a ErbB2 (v-erb-b2 avian erythroblastic leukemia viral oncogene homolog 2) interacting protein activity in cervical cancer, was reported to upsurge STAT3 phosphorylation, thus facilitating anoikis resistance and tumor invasiveness in vitro and in vivo (83). Here, ERBB2 is linked to neoplasm.