However, according to current evidence, the presence of intestinal anti-TG3 plasma cells seems not to consistently correlate with the level of serum TG3 antibodies (14), raising the possibility that two or more subsets of autoantibodies with different plasma cell origins may exist in DH, as suggested for TG2 autoantibody producing plasma cells in CeD (33). The gene discussed is TGM2; the disease is cranioectodermal dysplasia.