Naringin treatment also suppressed cell viability and proliferation, and promoted apoptosis in human small cell lung cancer cells (H69AR) by regulation of miR-126/Akt/mTOR/PI3K pathway via miR-126 overexpression and suppression of VCAM-1, p-Akt, PI3K, NF-κB, and p-mTOR pathways (Chen et al., 2018). The gene discussed is AKT1; the disease is small cell lung carcinoma.