In support, most of RBM47-dependent basal B-specific splicing events play well-known roles in cell-cell adhesion (CTNND1) [58], cytoskeleton organisation (ENAH, SLK, FNBP1) [59, 60], endocytosis (KIF13A, DNM2) [61] and association with the extracellular matrix (PLOD2) [62], which are all key processes for gaining the cell motility and invasiveness necessary in tumour metastasis (54–58). Here, RBM47 is linked to neoplasm.