Likewise, we observed multiple functional pathways (e.g., TRAIL [tumor necrosis factor-related apoptosis-inducing ligand] signaling and IL12 [interleukin-12]-mediated signaling pathways), biological processes (e.g., endoderm formation, response to oxygen radical), and phenotypes (e.g., arteriosclerosis, abnormal mitochondrial crista morphology) enriched specifically in SARS-CoV-2. This evidence concerns the gene TNFSF10 and arteriosclerosis disorder.