CD274 and neoplasm: The high expression level of tumor-infiltrating lymphocytes (TILs), programmed death-ligand protein-1 (PD-L1), highly tumor mutational burden (TMB), microsatellite instability (MSI), and mis-match repair deficiency (MMR) are the features of TNBC [8], which may contribute to TNBC patients suitable and sensitive to immunotherapy.