GFI1B and acute myeloid leukemia: The study by Maiques-Diaz et al. [77] and Vinyard et al. [76] which is in agreement with [78] stated that AML survival was dependent on LSD1 interaction with GFI1 and GFI1b (transcriptional repressors) and that the enzymatic action of LSD1 was not beneficial to the AML cells.