The results, presented in Table 3, confirmed the association of STAT4 and KCNH5 with PBC; while the replication of STAT4 was expected given that its role is known in PBC, the strongest replication was at KCNH5 (P = 5.83 × 10−25), a new candidate gene that only RHM detected. This evidence concerns the gene STAT4 and primary biliary cholangitis.