Beyond aspirin sensitivity, our observations could also be influenced by changes in Fn7-1 gene expression in response to lower levels of aspirin, including downregulation of important virulence factors like fap2, required to mediate CRC tissue localization and anti-tumor immunity (4, 14), and fadA, encoding a fusobacterial adhesin that engages E-cadherin to promote Wnt/β‐catenin signaling and drive cancer cell proliferation (12). Here, CDH1 is linked to colorectal carcinoma.